by Thomas E. Levy, MD, JD
June 01,
2022
from
OMNS Website
As of the writing of this
article, multiple news reports have recently addressed the
occurrence of
monkeypox virus infections in humans.
In the current setting of
the entire planet dealing with
the COVID 'pandemic' over the last two
and a half years, fear is readily stoked that another 'pandemic' with
a virus that comes from the same family of viruses as smallpox could
be poised to inflict widespread suffering and death.
This article will present
the significant scientific data and literature surrounding monkeypox
infection in humans, which clearly demonstrates that the monkeypox
virus presents NO threat of a 'pandemic' or even a large epidemic.
Monkeypox
Characteristics
Although many have never heard of it until recently, monkeypox
infection is not the result of the emergence of a new virus.
Rather,
it was first identified in captive cynomolgus monkeys in Denmark in
1958... 1
The first documented
human infection was reported in 1970 in a 9-month-old in the Congo.
After clinically recovering from the infection and its associated
rash over a month-long period, this baby then contracted the measles
and died six days later. 2
While not found
exclusively in remote populations in Central and West Africa,
limited monkeypox outbreaks appear to have occurred most commonly in
such areas of the world, where advanced malnutrition can potentially
make some otherwise benign infections life-threatening. 3
This initial case also
serves to highlight the fact that underlying chronic malnutrition
with moderate to severely depleted vitamin and mineral stores in
those living in such remote areas of Africa literally sets the stage
for contracting any infectious disease.
Quickly contracting
measles upon the resolution of the monkeypox virus is the logical
result of such an advanced depletion of nutrients in the body.
The typical mild to
moderate clinical presentation of the measles can easily evolve into
a fatal infection when a chronic state of nutrient depletion is
still further depleted by a month-long bout with the monkeypox
virus.
Monkeypox cases have only occurred as very limited outbreaks, never
as an epidemic or a 'pandemic'.
Such an outbreak is a
cluster of cases in a given area from a pathogen with a limited
contagion risk. An epidemic/'pandemic' requires a pathogen that is
very easily spread.
This is not the case with
the monkeypox virus.
The United States has already had an outbreak
of monkeypox infection in 2003, involving 47 human cases felt to be
secondary to the importation of infected wild rodents from Ghana.
No secondary larger
outbreak or epidemic resulted, however.
Furthermore, no
human-to-human transmission was documented. 4
Typically, while
human-to-human transmission is certainly possible, it is the
exposure to and/or the consumption of infected animals, as well as
their consumption of each other, that both spreads this virus and
serves as a reservoir for it.
This is an additional
reason for its primary presence in Africa, in addition to the
overall poor nutrition on much of this continent. 5,6
Monkeypox is characterized as a zoonotic infection, meaning it can
transmit from animal to human, or vice-versa. 7
Asymptomatic monkeypox
infections are very common, as over half of the healthy persons in
an area of Ghana, which actually had no reported clinical human
cases of monkeypox at the time of this study, had positive
immunoglobulin G (IgG) antibodies against the monkeypox virus genus.
8
A similarly large
percentage of the healthy residents in a region of the Congo had
circulating antibodies as well. 9
Another study in Cameroon
found these antibodies in slightly over a third of the subjects
tested. 10
This indicates that
monkeypox is not typically severe
in its clinical course, much less fatal, in any human population...
And this would especially
be the case in the United States or in a comparable country with a
relatively high-quality level of nutrition as well as a relatively
widespread intake of vitamin and mineral supplementation.
Ebola, another virus that has been largely limited to African
countries, resulted in a substantial outbreak in West Africa from
2014 to 2016, but it never approached 'pandemic' or even significant
epidemic proportions.
Nevertheless, in the
nutrition-depleted populations in which it emerged, death resulted
in those individuals demonstrating clinical infection between 25%
and 90% of the time, enough to generate a great deal of fear that it
could spread and kill easily throughout the world. 11
And even though Ebola
killed many who became infected, a substantial number of those
individuals exposed developed natural immunity (IgG antibody)
response without ever becoming clinically ill.
Depending on the location
of the African community and the conditions of the testing protocol
itself,
up to 50% of exposed individuals, including those living
with clinically infected individuals, showed the development of
natural antibodies to Ebola without ever becoming ill. 12-18
And in spite of the
initial fear that was generated, no 'pandemic', epidemic, or even
minor outbreak of Ebola ever occurred in the United States, even
though international airline travel reliably introduced infected
individuals into the country. 19,20
Most of the fear currently seen with the potential spread of the
monkeypox virus is due to the fact that both monkeypox and smallpox
comes from the same genus of DNA viruses. 21
Smallpox has been
estimated to have killed between 300 and 500 million people in the
20th century... 22
Understandably, then,
anything that is remotely related to smallpox can be expected to
generate a great deal of concern.
While the smallpox vaccine is credited for the effective eradication
of smallpox, it is also believed by some that waning vaccine
immunity is currently leaving over 70% of the world's population
unprotected against smallpox, as this vaccine has not been routinely
administered since 1980. 23
Some estimates indicate
that the smallpox vaccination has offered roughly an 85% protection
against monkeypox infection. 24
And since the vaccine
immunity against smallpox is felt to be waning, the associated
cross-immunity against related viruses like monkeypox is felt to be
fading as well. 25
However, monkeypox is simply not smallpox.
The evidence presented
above indicates that many asymptomatic infections occur with monkeypox, and that it is far less contagious than smallpox, with
human-to-human transmission being decidedly uncommon.
Concurrent epidemics or
outbreaks of smallpox and monkeypox have not been reported, and
smallpox is not a zoonotic infection like monkeypox, but infects
humans only. 26
Finally, in the more well-fed and healthy populations in the world,
monkeypox is simply not a killer virus once contracted. The typical
clinical course of monkeypox in such populations much more resembles
chickenpox than smallpox.
Even if the presumed
waning protection of the old smallpox vaccinations results in some
increase in human monkeypox cases, it will not turn monkeypox into
the highly contagious and deadly killer that is smallpox.
Easily
Prevented, Readily Resolved
While some viruses are much more contagious and much more capable of
causing severe illness and even death than others, they all share
therapeutic susceptibilities.
As devastating as Ebola
has been to many of the individuals in Africa who have contracted
it, bio-oxidative treatment readily resolves it as well as any other
virus that is treated before too much advanced organ damage has
already taken place.
At the height of
the
Ebola scare in 2014, Drs. Robert Rowen and Howard Robins were so
convinced of their ability to cure Ebola infections that they put
themselves directly in harm's way by traveling to Sierra Leone, a
West African epicenter of Ebola infection at that time.
Of note, many physicians
and other healthcare providers in this area of Africa were dying
from the infection at that time.
The primary therapy they used to treat the Ebola patients was ozone.
And even though great local resistance was met in gaining access to
patients, four individuals were successfully treated with ozone
therapy.
The cornerstone ozone
application was direct intravenous ozone gas injection.
Supplemental oral vitamin
C therapy was administered as well to address its infection-induced
deficiency, to bolster immune function, and to minimize the impact
of any possible pro-oxidant Herxheimer-like rapid virus kill-off
reactions.
All four patients
improved immediately after the first treatment and complete
resolution of their infections was seen between two to five days.
Furthermore, no progression of any Ebola-related symptoms was seen
after the first ozone treatments were administered. 27
Other acute viral syndromes that have initially had much of the
world on edge in recent years have also been proven to be readily
curable, although not with any known prescription drugs.
In 2014,
Chikungunya
virus received a lot of attention, and some outbreaks with this
virus were sizeable, although never really reaching epidemic
proportions.
This viral infection
typically left those infected with debilitating symptoms, often
resulting in severe pain in many of the joints in the body.
The most immunocompetent
individuals would often resolve their most severe symptoms in about
a week, but in some the joint pain would become chronic and last as
long as five years.
Separate one-time
intravenous infusions with two bio-oxidative agents,
vitamin C and
hydrogen peroxide,
...in 56 patients were highly effective in both
completely resolving this viral infection, as well as in immediately
alleviating much of the chronic pain that remained long after the
acute phase of the infection. 28
Treatment with just
high-dose vitamin C intravenously (as much as 100 grams daily) in
the acute stage of viral infection with Chikungunya, influenza, Zika,
and dengue has also been reported to be similarly curative.
29-32
In a nutshell, unless the patient has advanced organ damage and is
very near death, intravenous vitamin C, in sufficient doses, can
always be expected to save the patient from succumbing to an
advanced infection, especially viral.
As the primary
electron-donating nutrient in the body, enough vitamin C must be
administered to both neutralize the new, ongoing infection-derived
pro-oxidants (toxins) while restoring (reducing) the physiological
function of those biomolecules that have already been oxidized.
A sizeable number of
integrative medicine practitioners who appreciate the therapeutic
value of IV vitamin C remain needlessly wary of 50- to 100-gram
infusions of vitamin C.
This unnecessary caution
too often results in a total daily dose of vitamin C of 25 grams or
less that proves insufficient to save the patient with severe and
widespread oxidative damage secondary to an advanced infection.
Nevertheless, even such lower doses can oftentimes suffice, just not
as reliably so.
The experience at the Riordan Clinic alone in Wichita, Kansas
clearly establishes the safety (and efficacy) of even the highest
dosing regimens of vitamin C on a routine basis.
Over the past 32 years,
over 150,000 intravenous infusions of vitamin C have been
administered at Riordan campuses.
Doses have varied from
7.5 to 250 grams daily, with 50 grams being the most common dose
administered. NO significant adverse side effects have occurred, and
NO kidney stones have resulted.
For more information on
the vitamin C-related research and results of the Riordan Clinic,
see:
https://riordanclinic.org/journal-articles/.
The primary bio-oxidative therapies (vitamin C, hydrogen peroxide,
ozone, ultraviolet blood irradiation, and hyperbaric oxygen) have
all been shown to eradicate any viral infections for which they have
been properly administered.
As noted above,
intravenous ozone can promptly resolve even an advanced viral
infection whenever access to it is available.
Properly-dosed
intravenous hydrogen peroxide is comparably effective, and as long
as the healthcare practitioner is willing to use it, its expense is
nominal and it is available literally everywhere.
Vitamin C, ultraviolet
blood irradiation, and hyperbaric oxygen therapy are incredibly
effective as well, but less available and anywhere from slightly to
substantially more expensive to apply than the hydrogen peroxide
and/or the ozone therapies.
These therapies, along
with other supportive antipathogenic measures, are discussed in
greater detail elsewhere. 33
Another great option for dealing with any virus once contracted is a
combination vitamin C-cortisol approach, especially when the
intravenous administration of bio-oxidative agents is not readily
available, if at all.
A sizeable oral dose of
vitamin C (3 to 5 grams, liposome-encapsulated or as sodium
ascorbate powder) along with 20 mg of cortisol (hydrocortisone) is
dramatically effective in its clinical impact, often resulting in a
prompt cessation of infection evolution followed shortly thereafter
by complete resolution.
As a very general
guideline, the vitamin C/cortisol should be taken three times daily
until baseline health is restored. Complete clinical resolution is
typically seen in 12 to 36 hours.
A more prolonged
treatment plan is only required when the pathogen has had a longer
time to replicate and clinical illness is more pronounced when
therapy is initiated. 34,35
Recap
Monkeypox virus should never be confused with smallpox, even though
the viruses have some common family roots.
Smallpox is a human
infection, and monkeypox is primarily limited to infections in
susceptible animal populations...
When monkeypox does
infect a human, its clinical course is little more than that of a
typical case of chickenpox, as long as the infected individual is
not grossly malnourished.
And even in populations
with significant nutrient depletion, monkeypox is very often a
completely asymptomatic infection, as high levels of protective
antibodies to monkeypox have been documented in significant
percentages of these populations.
Also, unlike smallpox,
monkeypox has both a very low level of contagion and only
rarely
results in a fatal outcome, even in the most susceptible of
populations.
A good level of nutrition, along with judicious supplementation with
vitamins and minerals, will almost completely prevent the
transmission of monkeypox, from either an infected animal or human.
And when it is
contracted, the application of any of a number of bio-oxidative and
other therapies will give a rapid resolution to this infection.
This ease of prevention
and susceptibility to rapid cure should be kept in mind before
deciding to proceed directly with any monkeypox vaccinations that
end up being offered to the public...
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34. Levy T
(2021)
http://orthomolecular.org/resources/omns/v17n28.shtml
35. Levy T
(2022)
http://orthomolecular.org/resources/omns/v18n06.shtml
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